Disease Model of GATA4 Mutation Reveals Transcription Factor Cooperativity in Human Cardiogenesis [ChIP-Seq]
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE85628
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资源简介：
Heterozygous mutations in GATA4 cause congenital heart defects and cardiomyopathy through unknown mechanisms. To gain insights into the genome-wide localization perturbations during human cardiac development due to GATA4 heterozygosity, we performed ChIP-seq of wildtype and GATA4-G296S diseased cardiomyocytes. ChIP sequencing of GATA4, TBX5, MED1, H3K4me3, H3K36me3, H3K27ac, H3K27me3, plus input, from 75 wildtype or diseased pluripotent stem cell derived cardiomyocytes
创建时间：
2019-05-15
