IL-10 production and metabolic regulation in macrophages are linked by a TLR-driven CREB-mediated mechanism

ChEBI2015-07-08 更新2026-07-12 收录

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巨噬细胞功能

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-64844 数据链接链接失效反馈

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IL-10 production and metabolic regulation in macrophages are linked by a TLR-driven CREB-mediated mechanism

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2015-07-08

Microbial Energy Metabolism Fuels a CSF2-dependent Intestinal Macrophage Niche within Solitary Isolated Lymphoid Tissues via purinergic signaling.. Microbial Energy Metabolism Fuels a CSF2-dependent Intestinal Macrophage Niche within Solitary Isolated Lymphoid Tissues via purinergic signaling.

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巨噬细胞生态位

Gene expression comparison across CD11b+ cells from Csf2-sufficient and deficient mice. Overall design: Steady state isolated CD11b+ cells from colon lamina propria or solitary isolated lymphoid tissu

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D- 2-hydroxyglutarate is an immunometabolite that accumulates in macrophages after TLR activation and inhibits inflammatory responses

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Macrophages undergo extensive metabolic rewiring upon activation and this serves many roles beyond the production of energy and anabolic building blocks. So-called immunometabolites that accumulate up

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DataSheet_1_Low-density lipoprotein balances T cell metabolism and enhances response to anti-PD-1 blockade in a HCT116 spheroid model.zip

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IntroductionThe discovery of immune checkpoints and the development of their specific inhibitors was acclaimed as a major breakthrough in cancer therapy. However, only a limited patient cohort shows s

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H3K27me3 occupancy in primary human peripheral blood derived- macrophages. H3K27me3 occupancy in primary human peripheral blood derived- macrophages

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Expression of HPGD in regulatory T cells prevents adipose tissue inflammation and metabolic dysfunction (data set 2)

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Regulatory T cells (Treg cells) are important for the prevention of autoimmunity and lately, their role in maintaining tissue homeostasis has been demonstrated. They exert their function via different

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IL-10 production and metabolic regulation in macrophages are linked by a TLR-driven CREB-mediated mechanism

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