We report the consequences of the disruption of the Fmo5 gene on transcription of genes in liver. Comparison of liver gene expression in wild-type and Fmo5(-/-) mice
aEyelid closure defects were observed in 100% of Dlgh-1−/− mice on the C57BL/6J background. However, the severity of the phenotype varied from animal to animal.bDefects in bone formation were observed
Wild type or Pax6 fx/fx; Le-Cre-positive (Pax6-/-) surface ectoderm from E9.5 mouse embryos was laser micodissected from three embryos of each genotype. Total RNA was purified, pooled for each genotyp
Cryosections of wild-type (A, C, E, G, I, K) and αA-CLEF (B, D, F, H, J) embryos stained for DAPI (C, D), for early differentiation marker Prox1 (A, B), Sox1 (E, F), for marker of fiber cell different
