Supp Table S3 from Targeting CCL2/CCR2 Signaling Overcomes MEK Inhibitor Resistance in Acute Myeloid Leukemia

Figshare2024-05-15 更新2026-04-28 收录

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https://figshare.com/articles/dataset/Supp_Table_S3_from_Targeting_CCL2_CCR2_Signaling_Overcomes_MEK_Inhibitor_Resistance_in_Acute_Myeloid_Leukemia/25826396 数据链接链接失效反馈

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RNA-seq gene list and pathway correlation with drug resistance

创建时间：

2024-05-15

Mus musculus Raw sequence reads

m6A甲基化

"RNA研究"

In order to further clarify the relationship between m6A methylation level and gene expression, we measured the level of mRNA changes in mouse with HFpEF using the RNA sequencing data of input experim

NIAID Data Ecosystem70

Reversing Melanoma Cross-Resistance to BRAF and MEK Inhibitors by Co-Targeting the AKT/mTOR Pathway

肿瘤耐药机制

信号通路靶向治疗

BackgroundThe sustained clinical activity of the BRAF inhibitor vemurafenib (PLX4032/RG7204) in patients with BRAFV600 mutant melanoma is limited primarily by the development of acquired resistance le

Figshare2016-01-18 更新50

SAKE (Single-cell RNA-Seq Analysis and Klustering Evaluation) Identifies Markers of Resistance to Targeted BRAF Inhibitors in Melanoma Cell Populations. Homo sapiens isolate:451Lu is a selected xenograft aggressive metastatic sub-line of WM164. WM164 is a metastatic human melanoma cell line established from a metastatic site (right upper arm) in a 22-year-old male with stage IV superficial spreading melanoma. 451Lu cell line features the specific V600E (Val600Glu) mutation at codon 600 in the BRAF gene.

单细胞转录组学

肿瘤耐药机制

The goal of this study is to apply the SAKE algorithms to identify drug-resistant cellular populations as human melanoma cells respond to targeted BRAF inhibitors. Single-cell RNA-Seq data from 10x Ge

NIAID Data Ecosystem20

Small RNA sequencing in normal and psoriatic skin. Homo sapiens

细胞miRNA表达谱

"RNA研究"

We report the application of Illumina small RNA sequencing to normal human skin, as well as uninvolved and involved psoriatic skin. By obtaining over 600 million qualified reads from 20 healthy contro

NIAID Data Ecosystem70

Suppression of adaptive responses to targeted cancer therapy by transcriptional repression [RNA-seq]. Homo sapiens

肿瘤耐药机制

转录组药物响应

Large-scale genomic profiling efforts have facilitated the characterization of molecular alterations in cancers and aided the development of targeted kinase inhibitors for a wide array of cancer types

NIAID Data Ecosystem50

Supp Table S3 from Targeting CCL2/CCR2 Signaling Overcomes MEK Inhibitor Resistance in Acute Myeloid Leukemia

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