Apolipoprotein E4 (APOE4) is the strongest known genetic risk factor for late-onset Alzheimer’s disease (AD). Conditions of stress or injury induce APOE expression within neurons, but the exact roles

NIAID Data Ecosystem50

The proto-oncogene DEK regulates neuronal excitability and tau accumulation in Alzheimer’s disease vulnerable neurons.. The proto-oncogene DEK regulates neuronal excitability and tau accumulation in Alzheimer’s disease vulnerable neurons.

阿尔茨海默病分子机制

tau蛋白病理

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem20

Additional file 1 of Tau interactome and RNA binding proteins in neurodegenerative diseases

tau蛋白病理

神经退行性疾病蛋白质

Additional file 1: Supplementary Table 1. List of post-translational modifications detected on tau across AD, AGD, CBD, PiD and PSP. Supplementary Table 2. Summary of human tau interactome and localis

Figshare2022-10-18 更新50

Data_Sheet_1_Regionally restricted modulation of Sam68 expression and Arhgef9 alternative splicing in the hippocampus of a murine model of multiple sclerosis.docx

神经退行性机制

突触功能障碍

Multiple sclerosis (MS) and its preclinical models are characterized by marked changes in neuroplasticity, including excitatory/inhibitory imbalance and synaptic dysfunction that are believed to under

NIAID Data Ecosystem40

Additional file 4 of Tau oligomers modulate synapse fate by eliciting progressive bipartite synapse dysregulation and synapse loss

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