E-MTAB-2973

NIAID Data Ecosystem2026-05-02 收录

多发性硬化症机制

运动干预转录组学

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Transcription profiling by array of whole blood from 7 healthy controls and 7 multiple sclerosis (MS) patients who improved their fatigue status after a program of physical exercise

创建时间：

2025-01-23

Supplementary Figure 1

急性肺损伤基因表达

运动干预转录组学

Supplementary Fig. 1. The heatmap and unsupervised hierarchical clustering show the gene expression profile of selected exercise-modulated acute lung injury (ALI) response genes in (A) t

DataCite Commons2025-06-01 更新70

Targeting lipophagy in macrophages improves repair in multiple sclerosis

多发性硬化症机制

吞噬与脂噬调控

Foamy macrophages containing abundant intracellular myelin remnants are an important pathological hallmark of multiple sclerosis. Reducing the intracellular lipid burden in foamy macrophages is consid

Taylor & Francis Group2022-11-02 更新30

Ursolic acid derivative UAOS-Na treats experimental autoimmune encephalomyelitis by immunoregulation and direct anti-demyelination

免疫性疾病药物研发

多发性硬化症机制

Multiple sclerosis (MS) is a chronic, inflammatory, and demyelinating disease of the central nervous system (CNS). Ursolic acid (UA) can be used in the MS treatment with anti-inflammatory and neuropro

NIAID Data Ecosystem20

Cladribine treatment specifically impairs peripheral blood memory B cell clones and clonal expansion in multiple sclerosis patients

多发性硬化症机制

细胞克隆动态

We performed next-generation sequencing of Ig heavy chain transcripts and Ig mass spectrometry in cladribine-treated patients with relapsing-remitting multiple sclerosis (n = 8) at baseline and after

NIAID Data Ecosystem40

Pregnancy changes expression in peripheral blood mononuclear cells of healthy donors. Homo sapiens

妊娠免疫调节

多发性硬化症机制

Background: pregnancy is associated with reduced activity of multiple sclerosis (MS). However, the biological mechanisms underlying this pregnancy-related decrease in disease activity are poorly under

NIAID Data Ecosystem30

E-MTAB-2973

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