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Transcriptome analysis of differentiating normal and leukemic erythroid progenitors

ChEBI2012-02-27 更新2026-07-21 收录

白血病分子机制

祖细胞分化

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-35384 数据链接链接失效反馈

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资源简介：

Transcriptome analysis of differentiating normal and leukemic erythroid progenitors

应用场景：

创建时间：

2012-02-27

白血病分子机制

ALOX5基因功能

The purpose of the study is to identify the role of ALOX5 in acute myeloid leukemia

NIAID Data Ecosystem50

LIN28B is overexpressed in specific subtypes of pediatric leukemia and regulates lncRNA H19 [K562]. Homo sapiens

白血病分子机制

LIN28B基因调控

The stem cell gene LIN28B was recently shown to be overexpressed in a foetal-like subgroup of juvenile myelomonocytic leukaemia. Given the involvement of LIN28B in a variety of solid paediatric cancer

NIAID Data Ecosystem10

TCF3::HLF orchestrates an enhancer-promoter network with activation of MEF2C to promote an immature HSC gene expression program in leukemia.

白血病分子机制

基因组调控

Oncogenic fusion transcription factors (TFs) frequently drive hematopoietic malignancies by altering gene expression in key developmental programs. TCF3::HLF is a fusion TF that characterizes a rare,

NIAID Data Ecosystem00

ZFP36L2 post-transcriptionally controls acute myeloid leukemia differentiation (MOLM13 HiC). ZFP36L2 post-transcriptionally controls acute myeloid leukemia differentiation (MOLM13 HiC)

白血病分子机制

三维基因组学

Genome-wide assessment of TADs in AML cells Overall design: TAD domains in MOLM13 AML cells

NIAID Data Ecosystem40

Transcription profiling of mouse leukemia (HSC) haematopoietic stem cells expressing BCR/ABL

白血病分子机制

干细胞转录组

Transcription profiling of mouse leukemia (HSC) haematopoietic stem cells expressing BCR/ABL

ChEBI2008-04-04 更新00