首页/数据集/Transcriptional profiling of Hutchinson-Gilford Progeria syndrome fibroblasts reveals deficits in mesenchymal stem cell commitment to differentiation related to early events in endochondral ossification.

Transcriptional profiling of Hutchinson-Gilford Progeria syndrome fibroblasts reveals deficits in mesenchymal stem cell commitment to differentiation related to early events in endochondral ossification.

NIAID Data Ecosystem2026-03-14 收录

早衰症分子机制

干细胞分化

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE206707 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

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创建时间：

2022-12-01

早衰症分子机制

miRNA表达谱分析

We first examined changes of miRNAs and expression using microarrays ( Exiqon miRCURY™ microarray LNA v.18.0) of GCs from 10 bPOI and 10 age and BMI matched normal control women. Seventy-five miRNAs (

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Characterization of cardiosphere-derived cells.

心脏球衍生细胞

干细胞分化

CDCs were isolated from cardiac tissue explants of healthy pigs. The figure A shows explants in culture with some fibroblast-like cells migrating from them (A.I), cardiospheres with CDCs migrating fro

NIAID Data Ecosystem20

Generating human artery and vein cells from pluripotent stem cell highlights the arterial tropism of Nipah and Hendra viruses. Homo sapiens

干细胞分化

病毒趋向性分析

Stem cell research endeavors to generate specific subtypes of classically-defined cell-types. Here we generate >90% pure human artery or vein endothelial cells from pluripotent stem cells within 3- 4

NIAID Data Ecosystem10

Cardiac differentiation of embryonic stem cells recapitulates embryonic cardiac development.. Mus musculus

心脏发育

干细胞分化

Mouse embryonic stem cells can differentiate in vitro into spontaneously contracting cardiomyocytes. The main objective of this study was to investigate cardiogenesis in cultures of differentiating em

NIAID Data Ecosystem20

Original data of "CCL3 in bone marrow microenvironment causes trabecular bone loss and bone marrow adiposity via triggering the imbalance between osteogenic and adipogenic differentiation of BMSCs in aged mice"

衰老代谢机制

干细胞分化

Original data of "CCL3 in bone marrow microenvironment causes trabecular bone loss and bone marrow adiposity via triggering the imbalance between osteogenic and adipogenic differentiation of BMSCs in

NIAID Data Ecosystem10

Transcriptional profiling of Hutchinson-Gilford Progeria syndrome fibroblasts reveals deficits in mesenchymal stem cell commitment to differentiation related to early events in endochondral ossification.

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