RNA-seq on aortic valves from WT vs N1-haploinsufficient telomere-shortened (generation 2 Terc-/-) mice with either healthy AVs or AV stenosis treated with control solvent or XCT790 Overall design: RN
To investigate the potential pathogenic role of SLC25A3 defects in myocardial hypertrophy, we performed RNA sequencing on WT and KO cell groups after culturing hiPSC-CMs.
