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Non-tumor/tumor intestinal tissue of control or intestine-specific HAI-1 deficient Apc(Min/+) mice

ChEBI2013-04-01 更新2026-07-21 收录

结直肠癌模型

基因功能缺失

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-40856 数据链接链接失效反馈

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资源简介：

Non-tumor/tumor intestinal tissue of control or intestine-specific HAI-1 deficient Apc(Min/+) mice

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创建时间：

2013-04-01

肿瘤治疗响应

基因功能缺失

We used CRISPR/Cas9 system to generate WT Control and RUNX1-KO MDS-L cells isolated as single clones from the parental MDS-L cell line. We have observed that RUNX1-KO MDS-L cells are resistant to Lena

NIAID Data Ecosystem100

Transcription profiling of HSCs from SYCNRIP WT and KO mice. Transcription profiling of HSCs from SYCNRIP WT and KO mice

干细胞转录组学

基因功能缺失

SYNCRIP depletion results in HSCs losing their self renewal abilities. Next generation sequencing was conducted in SYNCRIP WT and KO HSCs to investigate the transcriptional changes that occur, and res

NIAID Data Ecosystem60

Molecular Evaluation of Patient-Derived Colorectal Cancer Explants as a Pre-clinical Mouse Model of Colorectal Cancer

结直肠癌模型

个性化药物测试

Mouse models have been developed to investigate colorectal cancer etiology and evaluate new anti-cancer therapies. While genetically engineered and carcinogen-induced mouse models have provided impor

NIAID Data Ecosystem50

Whole-genome expression analysis of Rnaseh2b(E202X/E202X) embryos. Mus musculus

胚胎发育转录组学

基因功能缺失

The transcriptome of E9.5 embryos lacking functional RNase H2 (Rnaseh2bE202X/E202X) was compared to age-matched wild type controls to investigate the molecular basis of growth arrest in Rnaseh2-/- mic

NIAID Data Ecosystem90

Splicing Variation Contributes to Functional Dysregulation of Genes in Acute Myeloid Leukemia

AML可变剪接

基因功能缺失

Acute Myeloid Leukemia (AML) has been associated with somatic mutations in numerous genes; however, the penetrance of these mutations is low. Here we investigate the contribution of alternative splici

NIAID Data Ecosystem60

Non-tumor/tumor intestinal tissue of control or intestine-specific HAI-1 deficient Apc(Min/+) mice

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