Mechanism of inert inflammation in an immune checkpoint blockade-resistant tumor subtype bearing transcription elongation defects
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE212182
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High baseline CTLs in TEdef+ tumors result from the aberrant activation of the Nucleic Acid Sensing-TBK1-CCL5/CXCL9 signaling cascade, which induces an immunosuppressive microenvironment with increased regulatory T cells and exhausted CTLs. 2 Control, 2 Flavopiridol teated
创建时间：
2022-09-06
