autr: Viruses obtained from Flu PCR amplicons lacking a t1 termination signal. bUTR: No viruses obtained when Flu PCR amplicons lacked the pol 1 promoter sequence.
Competition and interplay between host and viral gene expression programs is a critical determinant of infection susceptibility and a potential target for anti-viral therapies. Viral infection is well
(C) MDCK cells were infected at MOI 0.05 with the following viruses: WT PR8 and PR8-PA(∆X) strains with no insert, PR8 with WT or mutant STOML2 sequences inserted in the WT PR8 or PR8-PA(∆X) backgrou
aVirus titers were determined by plaque assays of culture supernatants harvested at 24 h post-transfection and normalized to the pfu/ml value of the wt control, which was set at 1. bEAV genomic RNA le
