T helper cell type 2 ChIP-seq

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https://www.ncbi.nlm.nih.gov/sra/ERP105570

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CD4+ T cells were extracted from mouse and human. They were activated in vitro with CD3/28 and cultured with Il4. ChIPmentation was then performed

创建时间：

2023-10-13

T helper cell type 2 ChIP-seq. T helper cell type 2 ChIP-seq

CD4+ T cells were extracted from mouse and human. They were activated in vitro with CD3/28 and cultured with Il4. ChIPmentation was then performed

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ChIP-seq of Mus musculus T helper cell type 2 with retroviral overexpression of Bhlhe40 and Pparg. ChIP-seq of Mus musculus T helper cell type 2 with retroviral overexpression of Bhlhe40 and Pparg

T cells were extracted from spleens of CBLA mice and activated in vitro with CD3/28 on day 0. They were spinoculated with retroviruses overexpressing a 3xFLAG-gene of interest on day 1. On day 4, BFP-

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ChIP-seq of Mus musculus T helper cell type 2 with retroviral overexpression of Bhlhe40 and Pparg

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Tet2-mediated epigenetic programing of T follicular helper cell differentiation (ChIP-Seq). Tet2-mediated epigenetic programing of T follicular helper cell differentiation (ChIP-Seq)

Tet2-mediated demethylation is a key component of epigenetic programing that promotes lineage specific gene expression and contributes to cellular differentiation and function. While the differentiati

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RORa enforces stability of the T-helper-17 cell effector program (ChIP-Seq (2)). RORa enforces stability of the T-helper-17 cell effector program (ChIP-Seq (2))

Chronic inflammation is responsible for a number of debilitating human diseases including inflammatory bowel disease, multiple sclerosis, and rheumatoid arthritis. The Th17 subset of T lymphocytes is

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