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Association of single nucleotide variants with the presence of paracoccidioidomycosis (PCM), according to the studies retrieved in the systematic review.

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Figshare2026-03-19 更新2026-04-28 收录
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https://figshare.com/articles/dataset/_p_Association_of_single_nucleotide_variants_with_the_presence_of_paracoccidioidomycosis_PCM_according_to_the_studies_retrieved_in_the_systematic_review_p_/31814954
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The table presents the genetic models tested (codominant, dominant, recessive, overdominant, and log-additive), the genotype frequencies in healthy individuals and PCM patients, the odds ratios (OR) with 95% confidence intervals, p-values (including FDR correction), as well as the information criteria (AIC and BIC). Deviations from Hardy–Weinberg equilibrium (HWE) are highlighted in bold and in blue. Significant associations are highlighted in bold and in red (susceptibility). The best inheritance model, according to AIC and/or BIC, is highlighted in yellow. CTLA4: cytotoxic T-lymphocyte associated protein 4; CD209: CD209 molecule; FCGR2A: Fc gamma receptor IIa; IFNG: interferon gamma; IL10: interleukin 10; IL12A: interleukin 12A; IL12B: interleukin 12B; IL12RB1: interleukin 12 receptor subunit beta 1; IL18: interleukin 18; IL4: interleukin 4; JAK1: Janus kinase 1; TNF: tumor necrosis factor; VDR: vitamin D receptor. (XLSX)
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2026-03-19
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