INSIG1 in NASH pathophysiology and in the hepatic wound-healing response
收藏官方服务：
资源简介：
INSIG1 in NASH pathophysiology and in the hepatic wound-healing response
应用场景：
创建时间：
2021-04-14
相关数据集
Long-term metabolic effects of malnutrition: Liver steatosis and insulin resistance following early-life protein restriction
Early postnatal-life malnutrition remains prevalent globally, and about 45% of all child deaths are linked to malnutrition. It is not clear whether survivors of childhood malnutrition suffer from long
NIAID Data Ecosystem70
Deficiency in Gpr43 lead to exacerbation of NAFLD/NASH symptoms. Mus musculus strain:C57BL/6J
Non-alcoholic liver disease (NAFLD) is the hepatic manifestation of metabolic syndrome and can progress to non-alcoholic steatohepatitis (NASH). Alterations in the gut microbiome have been implicated
NIAID Data Ecosystem60
DataSheet_1_Gut-Derived Serotonin Contributes to the Progression of Non-Alcoholic Steatohepatitis via the Liver HTR2A/PPARγ2 Pathway.zip
The precipitous increase in occurrence of non-alcoholic steatohepatitis (NASH) is a serious threat to public health worldwide. The pathogenesis of NASH has not yet been thoroughly studied. We aimed to
NIAID Data Ecosystem20
Hepatic transcriptome analysis of PRDX1fl/fl and Alb-Cre；PRDX1fl/fl mice on MCD. Hepatic transcriptome analysis of PRDX1fl/fl and Alb-Cre；PRDX1fl/fl mice on MCD
The etiology of non-alcoholic steatohepatitis (NASH) has been complicated. An increasing body of literature has indicated that hepatic oxidative stress exerts a causal role in driving NASH. Neverthele
NIAID Data Ecosystem70
A maternal high-fat diet induces fetal origins of NASH-HCC in mice
Maternal overnutrition affects offspring susceptibility to nonalcoholic steatohepatitis (NASH). Male offspring from high-fat diet (HFD)-fed dams developed a severe form of NASH, leading to highly vasc
NIAID Data Ecosystem30
