首页/数据集/MOESM7 of MYCN and PRC1 cooperatively repress docosahexaenoic acid synthesis in neuroblastoma via ELOVL2

MOESM7 of MYCN and PRC1 cooperatively repress docosahexaenoic acid synthesis in neuroblastoma via ELOVL2

NIAID Data Ecosystem2026-03-11 收录

数据链接：

https://figshare.com/articles/dataset/MOESM7_of_MYCN_and_PRC1_cooperatively_repress_docosahexaenoic_acid_synthesis_in_neuroblastoma_via_ELOVL2/11414907 数据链接链接失效反馈

官方服务：

资源简介：

Additional file 7: Table S1. Patient information of tissue chip.

应用场景：

创建时间：

2019-12-19

神经母细胞瘤分子机制

miRNA生物标志物

To elucidate potential role of piRNAs in Neuroblastoma (NB), we performed the genome wide profiling in two human NB cell lines, IMR-32 and SH-SY-5Y by adopting high-throughput RNA sequencing (RNA-Seq)

NIAID Data Ecosystem30

Additional file 10 of EIF5A2 specifically regulates the transcription of aging-related genes in human neuroblastoma cells

衰老相关基因

神经母细胞瘤分子机制

Additional file 10. Correlation analysis between differentially expressed TFs and the validated UPR-related genes.

Figshare2023-02-07 更新20

PHOX2B promotes the occurrence and development of neuroblastoma by activating the MAPK signaling pathway. PHOX2B promotes the occurrence and development of neuroblastoma by activating the MAPK signaling pathway

神经母细胞瘤分子机制

MAPK信号通路

Studies have shown that PHOX2B is the first predisposition gene identified in neuroblastoma.To explore PHOX2B-dependent gene regulation, RNA-seq analysis was performed. Compared with the control group

NIAID Data Ecosystem20

Proteomics of NGF-TrkA signaling dynamics in neuroblastoma cells

生长激素信号通路

神经母细胞瘤分子机制

We used quantitative mass spectrometry-based proteomics to unravel global nerve growth factor (NGF)-induced TrkA signaling dynamics at the interactome, phosphoproteome and proteome level. A tetracycli

NIAID Data Ecosystem30

The ALK downregulated target gene HBP1 and repressor of MYCN activity as synergistic target for combined PI3K/HDAC inhibition [microarray]

神经母细胞瘤分子机制

基因表达调控

ALK mutations occur in 10% of primary neuroblastoma and represent a major target for precision treatment. In combination with MYCN amplification, ALK mutations infer an ultra-high-risk phenotype with

NIAID Data Ecosystem20

MOESM7 of MYCN and PRC1 cooperatively repress docosahexaenoic acid synthesis in neuroblastoma via ELOVL2

资源简介：

相关数据集