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Passage through the mammalian gut triggers a phenotypic switch required for Candida albicans commensalism

ChEBI2013-07-28 更新2026-07-21 收录

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肠道微生物定植

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-43972 数据链接链接失效反馈

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Passage through the mammalian gut triggers a phenotypic switch required for Candida albicans commensalism

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2013-07-28

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Macrophages may induce fungal apoptosis to fight against C. albicans, as previously hypothesized by our group. To confirm this hypothesis, we analyzed proteins from C. albicans cells after 3 h of inte

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Expression data from C. elegans. Caenorhabditis elegans

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We exposed wild-type Vibrio cholerae E7496, multiple Vibrio cholerae virulence factor deleted genes with intact hemolysin A gene [CVD109] and without hemolysin A gene [CVD110] in E7946, and E.coli OP5

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Crystal structure of NN domain of resistance protein

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Crystal structure of NN domain of resistance protein Descriptor: Tm-1 protein Authors: Matsumura, H, Katoh, E. Deposit date: 2014-02-27 Release date: 2014-08-13 Last modified: 2024-03-20 Method: X-RAY

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Transcriptome and proteome analysis of Salmonella enterica serovar Typhimurium systemic infection of wild type and immune-deficient mice

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Salmonella enterica are a threat to public health. Current vaccines are not fully effective. The ability to grow in infected tissues within phagocytes is required for S. enterica virulence in systemic

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Passage through the mammalian gut triggers a phenotypic switch required for Candida albicans commensalism

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