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Chromatin immunoprecipitation and sequencing (ChIP-Seq) in the murine leukemic cell lines Da-1
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创建时间：
2018-09-04
相关数据集
BET inhibition releases the Mediator complex from specific cis elements in acute myeloid leukemia cells (ChIP-Seq II)
Genome occupancy profiling by high throughput sequencing ChIP-seq of Brd4 with or without Doxycycline treatment to induce Med12 or Med23 shRNAs in MLL-AF9 transformed acute myeloid leukemia cells (RN2
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Preservation of MYBBP1A at the chromatin in MYBBP1A hemizygous cells
MYB Binding: Protein 1 A (MYBBP1A) is hemizygous in a majority of PDAC due to chromosome 17p deletion which also affects TP53. We generated isogenic homozygous and hemizygous MYBBP1A knockout cel
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Elevated enhancer-oncogene contacts and higher oncogene expression levels by recurrent CTCF inactivating mutations in acute T cell leukemia [4C]
CCCTC-binding factor (CTCF) regulates the 3D chromatin architecture by facilitating chromosomal loops. In addition to insulation of euchromatin from heterochromatin, CTCF is an important transcription
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Unraveling MLL1-fusion Leukemia: Epigenetic Revelations from an iPS Cell Point Mutation
Human iPS cells (WT ASE9203 cells) were obtained from Applied Stem Cell (ASC), and CRISPR-Cas9-mediated gene editing was completed by ASC using their proprietary CRISPR-Cas9 protocol to introduce the
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Homer peak annotations of soverlap with genomic features.
This table contains the fraction of peaks (per DBP) overlapping several genomic features. (CSV)
NIAID Data Ecosystem50
