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Supplementary files for Morphea & Tofacitinib
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DataSheet_1_Oxidative Stress-Mediated YAP Dysregulation Contributes to the Pathogenesis of Pemphigus Vulgaris.pdf
Pemphigus Vulgaris (PV) is a life-threatening autoimmune disease manifested with blisters in the skin and mucosa and caused by autoantibodies against adhesion protein desmoglein-3 (Dsg3) expressed in
NIAID Data Ecosystem120
Supplemental Tables for: Associations of pemphigus or pemphigoid with autoimmune disorders in US adult inpatients
Supplemental Tables 1-16 for "Associations of pemphigus or pemphigoid with autoimmune disorders in US adult inpatients" published in the Journal of the American Academy of Dermatology
Mendeley Data20
scRNA for pansclerotic morphea lesional
We compared the PSM lesional/non-lesional skin samples with the control skin in the analysis. lesional skin and non-lesional skin from each of the two PSM patients
NIAID Data Ecosystem10
Table_2_Alterations of Total Serum Immunoglobulin Concentrations in Pemphigus and Pemphigoid: Selected IgG2 Deficiency in Bullous Pemphigoid.docx
Pemphigus and pemphigoid diseases are organ-specific autoimmune diseases of the skin and/or mucous membranes, which are caused by autoantibodies targeting structural proteins of the skin. In other aut
NIAID Data Ecosystem80
Preclinical Studies Identify Non-Apoptotic Low-Level Caspase-3 as Therapeutic Target in Pemphigus Vulgaris
The majority of pemphigus vulgaris (PV) patients suffer from a live-threatening loss of intercellular adhesion between keratinocytes (acantholysis). The disease is caused by auto-antibodies that bind
Figshare2016-01-15 更新20
