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资源简介：
APP/Aβ mutants used in this study.
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创建时间：
2020-09-23
相关数据集
Alzheimer's disease. Alzheimer's disease
ES-derived neural cells Overall design: Alzheimer's disease vs. healthy controls
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The cryo-EM structure of AV-45 bound type3 amyloid beta 42 fibril.
The cryo-EM structure of AV-45 bound type3 amyloid beta 42 fibril. Descriptor: P3(40) Authors: Zhao, Q.Y, Tao, Y.Q, Liu, C, Li, D. Deposit date: 2023-08-15 Release date: 2024-08-21 Last modified: 2025
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Table_1_Benign Regulation of the Astrocytic Phospholipase A2-Arachidonic Acid Pathway: The Underlying Mechanism of the Beneficial Effects of Manual Acupuncture on CBF.DOC
BackgroundThe astrocytic phospholipase A2 (PLA2)-arachidonic acid (AA) pathway is crucial in understanding the reduction of cerebral blood flow (CBF) prior to cognitive deterioration. In complementary
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source data for fig5bcd
RyanR2-E4872Q (EQ) mutation promotes APP degradation through autophagic pathway
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Testosterone reduces neuronal secretion of Alzheimer's β-amyloid peptides
Alzheimer's disease (AD) is characterized by the age-related deposition of β-amyloid (Aβ) 40/42 peptide aggregates in vulnerable brain regions. Multiple levels of evidence implicate a central role for
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