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LNCaP cells in response to DHT stimulation and inhibition/knockdown of TOPO2B

ChEBI2010-07-29 更新2026-07-21 收录

前列腺癌分子机制

基因功能敲低

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-19445 数据链接链接失效反馈

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资源简介：

LNCaP cells in response to DHT stimulation and inhibition/knockdown of TOPO2B

应用场景：

创建时间：

2010-07-29

前列腺癌分子机制

基因表达调控

The goals of this study is to find effect of overexpression-TFEB in prostate cancer cell (DU145 cell line). Overall design: To further explore the potential molecular mechanisms of how TFEN affects ca

NIAID Data Ecosystem80

Results of morpholino microinjection experiments (2 experiments for each combination).

基因功能敲低

发育生物学实验

Results of morpholino microinjection experiments (2 experiments for each combination).

NIAID Data Ecosystem40

Rarg-Foxa1 signaling promotes luminal identity in prostate progenitors and is disrupted in prostate cancer

前列腺癌分子机制

FOXA1信号通路

Retinoic Acid is a crucial instructive signal for glandular identity through the transcriptional control of the pioneering factor FOXA1 in adult prostate progenitors.

NIAID Data Ecosystem10

Constitutively Active Androgen Receptor Variants Upregulate Expression of Mesenchymal Markers in Prostate Cancer Cells

前列腺癌分子机制

激素受体功能

Androgen receptor (AR) signaling pathway remains the foremost target of novel therapeutics for castration-resistant prostate cancer (CRPC). However, the expression of constitutively active AR variants

NIAID Data Ecosystem60

CENPA-Bound Genes and Transcriptional Profiling of CENPA-Depleted Prostate Cancer Cells

前列腺癌分子机制

CENP-A功能研究

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem30