miR-99 regulates normal and malignant hematopoietic stem cell self-renewal

NIAID Data Ecosystem2026-03-12 收录

造血干细胞调控

MicroRNA功能研究

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE101958 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

创建时间：

2021-07-25

RNA-sequencing of primary mouse hematopoietic stem progenitors

造血干细胞调控

干细胞多组学分析

Mechanisms of translational regulation are poorly understood in somatic stem cells. We used polysome profiling, RNA-sequencing, and proteomic approaches to examine translational regulation in hematopo

NIAID Data Ecosystem30

Hematopoietic stem cells from mice treated with G-CSF or saline alone for 7 days. Mus musculus

造血干细胞调控

TLR信号通路与炎症响应

Recent studies demonstratethat inflammatory signals regulate hematopoietic stem cells (HSCs). Granulocyte-colony stimulating factor (G-CSF) is often induced with infection and plays a key role in the

NIAID Data Ecosystem20

mRNA gene expression profile analysis in the CD4+ T cells with miR-7 deficiency. mRNA gene expression profile analysis in the CD4+ T cells with miR-7 deficiency

细胞基因表达

MicroRNA功能研究

To detect the mRNA gene expression profile in the unactivated and activated CD4+ T cells with miR-7 deficiency. Overall design: In this study, the total RNA was extracted from naive CD4+ T cells with

NIAID Data Ecosystem20

Expression analysis of miR-499 transgenic heart. Mus musculus

器官基因调控

MicroRNA功能研究

microRNAs are small non-coding RNAs that can affect gene expression. We used microarrays to analyze gene expression in miR-499 transgenic mouse hearts. Overall design: Cardiac ventricles were collecte

NIAID Data Ecosystem20

Transcriptomic profiling of HeLa cells treated with miR-744-5p and control-miR. Homo sapiens

转录组学分析

MicroRNA功能研究

To have a global picture of the targets of the mir-744-5p, we assessed transcriptome changes, by deep-sequencing, of HeLa cells transfected with this miRNA or a control miRNA (cel-miR-231). Overall de

NIAID Data Ecosystem20

miR-99 regulates normal and malignant hematopoietic stem cell self-renewal

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