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Transcriptional profiling of human bronchial epithelial cell BEAS-2B exposed to diesel and biomass ultrafine particles

ChEBI2018-05-09 更新2026-07-12 收录

空气污染毒理学

呼吸道转录组学

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-5157 数据链接链接失效反馈

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资源简介：

Transcriptional profiling of human bronchial epithelial cell BEAS-2B exposed to diesel and biomass ultrafine particles

应用场景：

创建时间：

2018-05-09

宿主-病毒互作

呼吸道转录组学

As a means to identify host transcriptional responses to SARS-CoV-2 infection, we generated RNA-seq data on the human nose organoid model HNO2043. We sampled HNO204 cells infected with SARS-CoV-2 and

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Identification of 3 Endotypes In Pediatric ARDS Using Nasal Transcriptomics-Standard Input. Identification of 3 Endotypes In Pediatric ARDS Using Nasal Transcriptomics-Standard Input

儿科ARDS内型分型

呼吸道转录组学

This study used nasal transcriptomic profiling of the inferior turbinate in control and pediatric ARDS subjects to identify endotypes. This data set is for non-amplified specimens. The study identfied

NIAID Data Ecosystem60

Additional file 7: of Transcriptional profiling of human bronchial epithelial cell BEAS-2B exposed to diesel and biomass ultrafine particles

空气污染毒理学

差异基因表达分析

Table S4. Fold change of genes differentially expressed by the exposure to both diesel and biomass UFP. (XLS 67Â kb)

NIAID Data Ecosystem40

Type I interferon response in human HTBE and A549 cells. Type I interferon response in human HTBE and A549 cells

干扰素反应

呼吸道转录组学

RNA-seq was used to profile gene expression changes in human tracheobronchial epithelial (HTBE) and A549 cells following type I interferon treatment. Overall design: Transcriptomic comparison of type

NIAID Data Ecosystem60

Transcriptomic analysis of of SNX-5422-treated human primary tracheobronchial epithelial (TBE) cells.. Transcriptomic analysis of of SNX-5422-treated human primary tracheobronchial epithelial (TBE) cells.

抗病毒药物机制

呼吸道转录组学

Purpose: In addition to developing antivirals targeting specific SARS-CoV-2 viral proteins, there has been a growing effort in developing therapeutics targeting host proteins exploited by SARS-CoV-2 f

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Transcriptional profiling of human bronchial epithelial cell BEAS-2B exposed to diesel and biomass ultrafine particles

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