Granulocytic Ehrlichiosis in Mice Deficient in Phagocyte Oxidase or Inducible Nitric Oxide Synthase
收藏PubMed Central2026-05-16 收录
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https://pmc.ncbi.nlm.nih.gov/articles/PMC101771/
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资源简介：
Mice deficient in phox (gp91(phox−/−)) or NOS2 (NOS2(−/−)) were infected with the agent of human granulocytic ehrlichiosis (HGE) to evaluate the importance of these pathways in the eradication of HGE bacteria. NOS2(−/−) mice had delayed clearance of the HGE agent in comparison to control or gp91(phox−/−) mice, suggesting that reactive nitrogen intermediates play a role in the early control of HGE.
提供机构：
American Society for Microbiology (ASM)
