首页/数据集/Unexpected cross-talk between the pathogen defense mechanisms of apoptosis, necroptosis, and pyroptosis.

Unexpected cross-talk between the pathogen defense mechanisms of apoptosis, necroptosis, and pyroptosis.

NIAID Data Ecosystem2026-03-11 收录

细胞死亡机制

病原体免疫应答

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE132134 数据链接链接失效反馈

官方服务：

资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2019-11-29

细胞死亡机制

缺血性脑卒中分子靶点

Inhibition or genetic deletion of poly(ADP-ribose) polymerase-1 (PARP-1) is profoundly protective against a number of toxic insults in many organ systems1,2. In the brain the molecular mechanisms unde

NIAID Data Ecosystem80

Caspase 3; the chelator treated groups o61–65 and o66–70 contains significantly more apoptotic neurons than the remaining groups.

凋亡研究

细胞死亡机制

Caspase 3; the chelator treated groups o61–65 and o66–70 contains significantly more apoptotic neurons than the remaining groups.

NIAID Data Ecosystem60

Autophagy and Apoptosis Are Differentially Induced in Neurons and Astrocytes Treated with an In Vitro Mimic of the Ischemic Penumbra

缺血性卒中模型

细胞死亡机制

The development of clinical stroke therapies remains elusive. The neuroprotective efficacies of thousands of molecules and compounds have not yet been determined; however, screening large volumes of p

Figshare2016-01-19 更新40

Functional enrichment analysis results.

缺血再灌注损伤

细胞死亡机制

BackgroundIntestinal ischemia–reperfusion (II/R) injury is a severe clinical condition in which regulated cell death programs—including pyroptosis and necroptosis—have emerged as key drivers of tissue

Figshare2025-12-16 更新60

Casterton et al Figure 3 - part 3

细胞死亡机制

神经退行性疾病

Source data for Casterton et al. 2026 Karyoptosis mediates cell death and neurodegeneration upon proteotoxic stress. Figure 3 - part 3

Zenodo2026-05-05 更新50