Fetal brain response to maternal inflammation requires microglia
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2024-07-01
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Fetal brain response to maternal inflammation requires microglia [Maternal_Inflammation]. Fetal brain response to maternal inflammation requires microglia [Maternal_Inflammation]
In utero infection and maternal inflammation can adversely impact fetal brain development. Maternal systemic illness, even in the absence of direct fetal central nervous system infection, is associate
NIAID Data Ecosystem110
sj-xlsx-3-asn-10.1177_17590914221144549 - Supplemental material for Sex and APOE Genotype Alter the Basal and Induced Inflammatory States of Primary Astrocytes from Humanized Targeted Replacement Mice
Supplemental material, sj-xlsx-3-asn-10.1177_17590914221144549 for Sex and APOE Genotype Alter the Basal and Induced Inflammatory States of Primary Astrocytes from Humanized Targeted Replacement Mice
DataCite Commons2024-08-28 更新70
Excitatory Neuron-Derived Interleukin-34 Controls Cortical Developmental Microglia Function
Neuron-microglia interactions dictate the development of neuronal circuits in the brain. However, the factors that support and broadly regulate these processes across developmental stages are largely
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Astrocytic PERK deficiency drives prefrontal circuit dysfunction and depressive-like behaviors
Major depressive disorder (MDD) is associated with dysfunction in prefrontal cortex (PFC) circuits, yet the glial mechanisms underlying these abnormalities remain unclear. Here, downregulation of the
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C9orf72 deficiency promotes microglial mediated synaptic loss in aging and amyloid accumulation [scRNA-seq]. C9orf72 deficiency promotes microglial mediated synaptic loss in aging and amyloid accumulation [scRNA-seq]
C9orf72 repeat expansions cause inherited amyotrophic lateral sclerosis/frontotemporal dementia (ALS/FTD) and result in both loss of C9orf72 protein expression and production of potentially toxic RNA
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