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Mechanistic differences in neuropathic pain modalities revealed by correlating behavior with global expression profiling

NIAID Data Ecosystem2026-03-12 收录

神经病理机制

基因表达-行为关联

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE102937 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2021-07-25

神经病理机制

背根神经节基因表达

Chronic neuropathic pain is a major morbidity of neural injury, yet its mechanisms are incompletely understood. Hypersensitivity to previously non-noxious stimuli (allodynia) is a common symptom. Here

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ATP mediates neuropathic pain of neuromyelitis optica spectrum disorder via microglial activation. ATP mediates neuropathic pain of neuromyelitis optica spectrum disorder via microglial activation

神经病理机制

视神经脊髓炎谱系疾病

Intractable neuropathic pain is recognized as a common symptom of neuromyelitis optica spectrum disorder (NMOSD). However, the underlying mechanism of NMOSD pain remains to be elucidated. Here, we est

NIAID Data Ecosystem70

The role of m6A/m-RNA methylation in stress response regulation

表观转录组学

神经病理机制

N6-Methyladenosine (m6A) and N6,2'-O-dimethyladenosine (m6Am) are abundant mRNA modifications that regulate transcript processing and translation. The role of both, here termed m6A/m, in the stress re

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Transcriptomic profiling in mice with CB1 deletion in primary sensory neurons

神经病理机制

疼痛治疗靶点筛选

in this study, we investigated whether CB1R conditional knockout from peripheral sensory neurons (CB1cKO) in mice may present mechanical allodynia, thermal hyperalgesia and change transcriptome expres

NIAID Data Ecosystem70

Additional file 3 of Positive interaction between GPER and β-alanine in the dorsal root ganglion uncovers potential mechanisms: mediating continuous neuronal sensitization and neuroinflammation responses in neuropathic pain

神经病理机制

神经炎症与神经元敏化

Additional file 3: Table S2. Primers Information of qPCR Experiment.

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