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Transcription profiling of mouse laser dissected motor neurons from neuron and spinal cord smaples from SOD1 mice

ChEBI2008-06-13 更新2026-07-21 收录

运动神经元转录组

ALS疾病模型

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-5037 数据链接链接失效反馈

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Transcription profiling of mouse laser dissected motor neurons from neuron and spinal cord smaples from SOD1 mice

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创建时间：

2008-06-13

神经元研究

ALS疾病模型

Oculomotor neurons, which regulate eye movement, are resilient to degeneration in the lethal motor neuron disease amyotrophic lateral sclerosis (ALS). It would be highly advantageous if motor neuron r

干细胞与再生医学数据中心2022-02-20 更新100

RNAseq of fluidically isolated human and mouse motor neuron somas and axons in vitro

运动神经元转录组

肌萎缩侧索硬化症（ALS）研究

Spinal motor axons traverse large distances to innervate target muscle, and thus require local control of cellular events for proper functioning of the distal axon. To interrogate axon-specific proces

NIAID Data Ecosystem70

Cortical glia in SOD1(G93A) mice are subtly affected by ALS-like pathology

ALS疾病模型

神经胶质细胞

The role of glia in amyotrophic lateral sclerosis (ALS) is undeniable. Their disease-related activity has been extensively studied in the spinal cord, but only partly in the brain. We present herein a

NIAID Data Ecosystem50

total RNA-seq from motor neuron (ENCSR575MQY)

运动神经元转录组

RNA-seq表达分析

RNA-seq on human motor neuron cell lines For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for

NIAID Data Ecosystem70

In Vivo Imaging Reveals Distinct Inflammatory Activity of CNS Microglia versus PNS Macrophages in a Mouse Model for ALS

ALS疾病模型

神经炎症成像

Mutations in the enzyme superoxide dismutase-1 (SOD1) cause hereditary variants of the fatal motor neuronal disease Amyotrophic lateral sclerosis (ALS). Pathophysiology of the disease is non-cell-auto

NIAID Data Ecosystem50

Transcription profiling of mouse laser dissected motor neurons from neuron and spinal cord smaples from SOD1 mice

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