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Cancer cells reprogram to metastatic state through the acquisition of platelet mitochondria

NIAID Data Ecosystem2026-05-01 收录

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computer code of article"Cancer cells reprogram to metastatic state through the acquisition of platelet mitochondria"

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创建时间：

2023-08-29

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多组学整合分析

Additional file 2. Gene set characterization. Gene set characterization of the DE genes between early and late stages. The downregulated genes are significantly enriched for cancer-related gene signat

NIAID Data Ecosystem50

Flag-RBM47 HITS-CLIP in brain metastatic derivatives of MDA-MB-231

RNA结合蛋白靶标

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Brain metastatic breast cancer cells were subjected to HITS-CLIP to identify the targets of the RNA binding protein RBM47 MDA231-BrM2a is characterized in PMID: 19421193; Related data published toget

NIAID Data Ecosystem40

shRNAi profiling of human MDA-MB-231 cells expressing anti-p53 (shp53) short-hairpin RNAs treated with TFGbeta or control

癌症转移机制

转录组功能分析

TGFβ ligands act as tumor suppressors in early stage tumors but are paradoxically diverted into potent prometastatic factors in advanced cancers. The molecular nature of this switch remains enigmatic.

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NFIC1 suppresses migration and invasion of breast cancer cells through interferon-mediated Jak-STAT pathway

癌症转移机制

Jak-STAT信号通路

NFIC1, the longest isoform of NFIC, is essential for the regulation on spatiotemporal expressions of drug-metabolizing genes in liver. However, the role of NFIC1 in breast cancer is not clear. Here we

NIAID Data Ecosystem40

NAT10 is identified as a novel driver for brain metastasis

癌症转移机制

乳腺癌脑转移

Metastatic breast cancer remains a major cause of cancer related deaths in women and there are few effective therapies against this advanced disease. Using in vivo and in vitro genetic screens, we ide

NIAID Data Ecosystem30

Cancer cells reprogram to metastatic state through the acquisition of platelet mitochondria

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