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mTOR-mediated phosphorylation of VAMP8 prevents a starvation-induced VAMP8-SCFD1 interaction that promotes SNARE activity and autolysosome fusion

NIAID Data Ecosystem2026-03-13 收录

自噬调控

SNARE介导的膜融合

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https://www.omicsdi.org/dataset/pride/PXD024642 数据链接链接失效反馈

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mTOR-mediated phosphorylation of VAMP8 prevents a starvation-induced VAMP8-SCFD1 interaction that promotes SNARE activity and autolysosome fusion

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创建时间：

2022-02-17

视网膜新生血管

自噬调控

Data of western blot analysis. The raw materials of western blot of VEGF and LC3 in different groups. (XLS 1095 kb)

NIAID Data Ecosystem110

Silica nanoparticles induce autophagosome accumulation via activation of the EIF2AK3 and ATF6 UPR pathways in hepatocytes

纳米颗粒毒性

自噬调控

Autophagy dysfunction is a potential toxic effect of nanoparticles. Previous studies have indicated that silica nanoparticles (SiNPs) induce macroautophagy/autophagy dysfunction, while the precise mec

DataCite Commons2024-02-20 更新90

Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance. Transcriptional regulation of autophagy-lysosomal function in BRAF-driven melanoma progression and chemoresistance

黑色素瘤分子机制

自噬调控

To Study the role of mutant Braf V600E and ERK dependent phosphorylation of TFEB in regulation of autophagy and lysosome biogenesis in the context of melanoma growth and metastasis, we used whole gene

NIAID Data Ecosystem40

GLIPR2 is a negative regulator of autophagy and the BECN1-ATG14-containing phosphatidylinositol 3-kinase complex

自噬调控

分子生物学

A key mediator of macroautophagy/autophagy induction is the class III phosphatidylinositol 3-kinase complex I (PtdIns3K-C1) consisting of PIK3C3/VPS34, PIK3R4/VPS15, BECN1, and ATG14. Although several

DataCite Commons2024-02-12 更新90

A novel orally available seleno-purine molecule suppresses triple-negative breast cancer cell proliferation and progression to metastasis by inducing cytostatic autophagy

三阴性乳腺癌治疗

自噬调控

Patients with triple-negative breast cancer (TNBC) often have a poor prognosis largely due to lack of effective targeted therapy. Using a library of seleno-purines coupled to a high-throughput biochem

NIAID Data Ecosystem50

mTOR-mediated phosphorylation of VAMP8 prevents a starvation-induced VAMP8-SCFD1 interaction that promotes SNARE activity and autolysosome fusion

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