Sepsis decreases lung SVEP1 expression in a murine model

NIAID Data Ecosystem2026-03-14 收录

脓毒症基因表达

器官疾病分子机制

数据链接：

https://www.omicsdi.org/dataset/biostudies-other/S-BSST942 数据链接链接失效反馈

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资源简介：

Basic datas in murine model experiments for SVEP1 and sepsis

创建时间：

2023-01-04

Gene expression of alveolar macrophage in mice with AT2 cell-specific Cpt1a deletion

器官疾病分子机制

免疫细胞转录组学

Carnitine palmitoyltransferase 1a (CPT1a) is the key regulator of mitochondrial long-chain fatty acid beta-oxidation (LCFAO). However, the functional significance of AT2 cell-specific LCFAO at baselin

NIAID Data Ecosystem100

HDMA Fragment Files + Count Matrices Lung Part2

单细胞多组学

器官疾病分子机制

This is the ATAC fragment files and RNA count matrices for HDMA (Liu*, Jessa*, Kim*, Ng* et al, bioRxiv 2025)

NIAID Data Ecosystem90

Whole-genome gene expression profiles of non-tumorous human lung tissues: GRNG set

器官疾病分子机制

基因表达谱分析

We profiled genome-wide gene expression in non-tumorous human lung tissues. The overall goal of this project is to improve our molecular understanding of various lung diseases including lung cancer an

NIAID Data Ecosystem20

Chronic Lung Injury by Constitutive Expression of Activation-Induced Cytidine Deaminase Leads to Focal Mucous Cell Metaplasia and Cancer

器官疾病分子机制

宫颈癌前病变病理

Activation-induced cytidine deaminase (AID) is an enzyme required for antibody diversification, and it causes DNA mutations and strand breaks. Constitutive AID expression in mice invariably caused lun

NIAID Data Ecosystem90

Additional file 3 of Cell-specific expression of lung disease risk-related genes in the human small airway epithelium

器官疾病分子机制

细胞基因表达

Additional file 3.

Figshare2020-07-30 更新90