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Down-regulation of cholesterol biosynthesis in forebrains of ERCC1-deficient mice

ChEBI2011-08-15 更新2026-07-21 收录

神经退行性机制

大脑脂质代谢

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-31199 数据链接链接失效反馈

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资源简介：

Down-regulation of cholesterol biosynthesis in forebrains of ERCC1-deficient mice

应用场景：

创建时间：

2011-08-15

神经退行性机制

细胞应激通路

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Purkinje neurons with loss of STIM1 exhibit age-dependent changes in gene expression and synaptic components

钙信号调控

神经退行性机制

The Stromal interaction molecule 1 (STIM1) is an ER-Ca2+ sensor and an essential component of ER-Ca2+ store operated Ca2+entry (SOCE). Loss of STIM1 affects metabotropic Glutamate Receptor 1 (mGluR1)

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Additional file 3 of Single-cell RNA-seq reveals alterations in peripheral CX3CR1 and nonclassical monocytes in familial tauopathy

单细胞转录组学

神经退行性机制

Additional file 3: Table S3. RNA integrity number for PBMC samples.

Figshare2024-09-11 更新40

Misconnecting the dots: altered mitochondrial protein-protein interactions and their role in neurodegenerative disorders

蛋白质相互作用

神经退行性机制

Introduction: Mitochondria (mt) are protein-protein interaction (PPI) hubs in the cell where mt-localized and associated proteins interact in a fashion critical for cell fitness. Altered mtPPIs are li

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AAGGG repeat expansions trigger RFC1-independent synaptic dysregulation in human CANVAS Neurons. AAGGG repeat expansions trigger RFC1-independent synaptic dysregulation in human CANVAS Neurons

神经退行性机制

功能障碍研究

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

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