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Western blot images for Figure 5D: p62 expression

NIAID Data Ecosystem2026-05-02 收录

Westernblot蛋白分析

神经退行性分子机制

数据链接：

https://data.mendeley.com/datasets/2rfc95k22n 数据链接链接失效反馈

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资源简介：

n α-Syn-SY5Y cells, silencing of circ_0005654 and transfection with siRNA of the miR-588 inhibitor， P62 was reversed

应用场景：

创建时间：

2025-04-30

衰老相关蛋白

神经退行性分子机制

The attachment of Post-Translational Modifications (PTMs) to proteins plays key roles in the regulation of the activity and stability of various proteins. Here we utilized the nematode Caenorhabditis

NIAID Data Ecosystem40

TDP-43表观遗传学

神经退行性分子机制

This is the summarized excel file containing the original information for all experiment-based figures and supplementary figures in our manuscript "Age-related demethylation of the TDP-43 autoregulato

Figshare2021-01-27 更新10

Expanded gene expression changes in inducible TDP-43 flies.

神经退行性分子机制

TDP-43蛋白相关基因表达

Genes are from same experiment and analysis as in Table 3, with less stringent criteria to identify potentially important genes. nACR, nicotinic acetycholine receptor.

Figshare2015-12-02 更新10

Global gene profiling of (-)-Epigallocatechin-3-gallate (EGCG)-induced neuronal death. Mus musculus

神经退行性分子机制

药物神经保护

Alzheimer’s disease (AD) is the most common form of adult-onset dementia with severe intellectual deterioration and is characterised by the accumulation of the amyloid-β (Aβ) peptides and the presence

NIAID Data Ecosystem30

Additional file 2: Table S2. of Dioxins and related environmental contaminants increase TDP-43 levels

环境毒理学

神经退行性分子机制

(DOCX 28.9 kb)

Figshare2017-05-06 更新10