Implication of Long noncoding RNAs in the endothelial cell response to hypoxia I.

ChEBI2016-08-25 更新2026-07-12 收录

缺氧分子机制

内皮细胞lncRNA功能

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-76741 数据链接链接失效反馈

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资源简介：

Implication of Long noncoding RNAs in the endothelial cell response to hypoxia I.

创建时间：

2016-08-25

Correction: Suppression of Mitochondrial Electron Transport Chain Function in the Hypoxic Human Placenta: A Role for miRNA-210 and Protein Synthesis Inhibition

缺氧分子机制

线粒体功能调控

Correction: Suppression of Mitochondrial Electron Transport Chain Function in the Hypoxic Human Placenta: A Role for miRNA-210 and Protein Synthesis Inhibition

Figshare2016-01-18 更新60

Quantitative proteomic analysis of hypoxia induced primary kidney cells

慢性肾脏病蛋白质组学

缺氧分子机制

Chronic kidney disease (CKD) is characterized by a slow and gradual loss of kidney function, with glomerular filtration loss over months or years, inevitably leading to end-stage renal disease. The re

NIAID Data Ecosystem50

lncRNA SCIRT in hypoxia/reoxygenation.. lncRNA SCIRT in hypoxia/reoxygenation.

缺氧分子机制

非编码RNA功能

To know effects of long noncoding RNA (lncRNA) SCIRT in hypoxia/reoxygenation (H/R). We compared expression profiles of human umbilical vein endothelial cells (HUVECs) under normal condition, H/R cond

NIAID Data Ecosystem20

LncRNAs and CircRNAs Expression Profiles of Human Olfactory Mucosa Mesenchymal Stem Cells between Hypoxic and Normoxic Conditions

细胞RNA表达谱

缺氧分子机制

To explore the molecules that control different biological characteristics of OM-MSCs between hypoxic and normoxic conditions.

NIAID Data Ecosystem60

Dataset for: Novel lncRNA regulated by HIF-1 inhibits apoptotic cell death in the renal tubular epithelial cells under hypoxia.

缺氧分子机制

lncRNA调控细胞凋亡

Chronic tubulointerstitial hypoxia plays an important role as the final common pathway to end-stage renal disease. HIF-1 (hypoxia inducible factor-1) is a master transcriptional factor under hypoxia,

WILEY2017-04-18 更新40

Implication of Long noncoding RNAs in the endothelial cell response to hypoxia I.

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