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Protective Role for the Disulfide Isomerase PDIA3 in Methamphetamine Neurotoxicity

ChEBI2011-11-15 更新2026-07-21 收录

神经毒理学

药物滥用神经保护机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-33707 数据链接链接失效反馈

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资源简介：

Protective Role for the Disulfide Isomerase PDIA3 in Methamphetamine Neurotoxicity

应用场景：

创建时间：

2011-11-15

神经毒理学

多巴胺能系统

Aconitine is one of the main bioactive and toxic ingredients of Aconitum species. Increasingly, aconitine has been reported to induce neurotoxicity. However, whether aconitine has effects on the dopam

NIAID Data Ecosystem60

Chronic ethanol exposure results in time and brain-region dependent changes in gene coexpression networks.

神经毒理学

基因共表达网络

We examined global gene expression profiles in amygdala (AMY), nucleus accumbens (NAC), prefrontal cortex (PFC) and Liver of male C57BL/6J mice exposed to 4 cycles of chronic intermittent ethanol (CIE

NIAID Data Ecosystem40

Possible Role of the Glycogen Synthase Kinase-3 Signaling Pathway in Trimethyltin-Induced Hippocampal Neurodegeneration in Mice

神经毒理学

海马神经退行性疾病机制

Trimethyltin (TMT) is an organotin compound with potent neurotoxic effects characterized by neuronal destruction in selective regions, including the hippocampus. Glycogen synthase kinase-3 (GSK-3) reg

NIAID Data Ecosystem20

Figure 2

神经毒理学

药物成瘾分子机制

Methamphetamine-induced region-specific transcriptomic and epigenetic changes in the brain of male rats

NIAID Data Ecosystem40

Significantly 36 down-regulated and up-regulated genes of neuronal ontology in exposure to BPA.

神经毒理学

基因表达调控

Significantly 36 down-regulated and up-regulated genes of neuronal ontology in exposure to BPA.

Figshare2015-12-02 更新20