Post-transcriptional manipulation of TERC reverses molecular hallmarks of telomere disease

ChEBI2016-08-02 更新2026-07-12 收录

端粒疾病机制

基因转录调控

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-81507 数据链接链接失效反馈

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Post-transcriptional manipulation of TERC reverses molecular hallmarks of telomere disease

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2016-08-02

Rapid Changes in Transcription Profiles of the Plasmodium yoelii yir Multigene Family in Clonal Populations: Lack of Epigenetic Memory?

疟原虫抗原

基因转录调控

The pir multigene family, found in the genomes of Plasmodium vivax, P. knowlesi and the rodent malaria species, encode variant antigens that could be targets of the immune response. Individual parasit

NIAID Data Ecosystem90

Taxi knock out Transcriptome. Drosophila melanogaster

神经遗传学

基因转录调控

Maintaining taxi homeostasis in neurons is important for maintaining the neuromuscular crosstalk and flight function in Drosophila melanogaster. Transcriptome analysis of taxi show that Adar is one of

NIAID Data Ecosystem70

FTH1P3, a Novel H-Ferritin Pseudogene Transcriptionally Active, Is Ubiquitously Expressed and Regulated during Cell Differentiation

基因转录调控

细胞分化机制

Ferritin, the major iron storage protein, performs its essential functions in the cytoplasm, nucleus and mitochondria. The variable assembly of 24 subunits of the Heavy (H) and Light (L) type composes

NIAID Data Ecosystem40

TFIIS.h, a new target of p53, regulates transcription efficiency of pro-apoptotic bax gene

癌症分子机制

基因转录调控

Investigation of whole genome gene expression level changes in human colon cancer cell lines in response to p53 activation by Inauhzin (INZ), and identification of novel p53 target genes. Part of the

NIAID Data Ecosystem70

The IKAROS Interaction with a Complex Including Chromatin Remodeling and Transcription Elongation Activities Is Required for Hematopoiesis

基因转录调控

造血细胞分化

IKAROS is a critical regulator of hematopoietic cell fate and its dynamic expression pattern is required for proper hematopoiesis. In collaboration with the Nucleosome Remodeling and Deacetylase (NuRD

Figshare2016-01-15 更新60

Post-transcriptional manipulation of TERC reverses molecular hallmarks of telomere disease

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