Feedback amplication of fibrosis through matrix stiffening and COX-2 suppression

ChEBI2010-07-15 更新2026-07-21 收录

纤维化机制

细胞外基质

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-22011 数据链接链接失效反馈

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Feedback amplication of fibrosis through matrix stiffening and COX-2 suppression

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2010-07-15

matrisome expression matrix

细胞外基质

组织不溶性matrisome分析

Our current study will provide a low-cost, simple, reliable and effective pipeline for tissue insoluble matrisome analysis in the quest to comprehend ECM discovery proteomic studies.

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Different effects of rat interferon alpha, beta and gamma on rat hepatic stellate cell proliferation and activation

纤维化机制

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BACKGROUND: Liver fibrosis is the common sequel of chronic liver diseases. Recent studies have identified hepatic stellate cells as the primary cell type mediating hepatic fibrogenesis. It has been de

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Increased dermal collagen bundle alignment in systemic sclerosis is associated with a cell migration signature and role of Arhgdib in directed fibroblast migration on aligned ECMs

纤维化机制

胶原束排列-细胞迁移关联

Systemic sclerosis (SSc) is a devastating disease affecting the skin and internal organs. Dermal fibrosis manifests early and Modified Rodnan Skin Scores (MRSS) correlate with disease progression. Tra

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Additional file 1 of Shotgun proteomics of extracellular matrix in late senescent human dermal fibroblasts reveals a down-regulated fibronectin-centered network

细胞外基质

衰老蛋白质组学

Additional file 1: Table S1.

NIAID Data Ecosystem30

DataSheet4_Pharmacological mechanisms of Fuzheng Huayu formula for Aristolochic acid I–induced kidney fibrosis through network pharmacology.zip

纤维化机制

中药网络药理学

Renal fibrosis, characterized by the destruction of renal tubules and interstitial capillaries and the accumulation of extracellular matrix proteins, is a common outcome of chronic renal diseases and

NIAID Data Ecosystem10

Feedback amplication of fibrosis through matrix stiffening and COX-2 suppression

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