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Chronic kidney disease and splicing events
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创建时间：
2013-08-16
相关数据集
Gene expression counts from fibroblast, strand-specific, BCM UDN
File description: Gene-level counts using the gtf file from the release 34 of GENCODE https://www.gencodegenes.org/human/release_34 Split counts spanning from one exon to another using
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Epigenetically-controlled tumor antigens derived from splice junctions between exons and transposable elements [ChIP-seq]. Epigenetically-controlled tumor antigens derived from splice junctions between exons and transposable elements [ChIP-seq]
To analyze regulation of JET expression, we generated Setdb1-deficient MC38 and B16OVA cells, using lentivirus-based CRISPR/Cas9. Ctrl and KO cells lines were used to perform ChIP-seq against H3K9me3
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List of genes and isoforms derived from different alternative splicing variants in PAH and PHH.
Modification type: D, gain or loss of domain; A, activity; L, localization.
Figshare2015-12-02 更新20
Three way interaction of Asian, male and ACE D allele on all-cause CKD, diabetic nephropathy and non-diabetic nephropathy.
Depend variable: log odds ratio of ACE I/D and CKD using allele type model. β: coefficients in meta-regression; se: standard error of β. Model 1: Hypertension was not included in independent variables
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Comparative sequence analysis of LRP1 and its truncated splice variant smLRP1.
Comparative sequence analysis of LRP1 and its truncated splice variant smLRP1.
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