Knocking out C9ORF72 exacerbates axonal trafficking defects associated with hexanucleotide repeat expansion and reduces levels of heat shock proteins II
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE143744
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资源简介：
wild type iPSCs were edited using CRISPR/Cas9 to knockout C9ORF72 followed by differentiation into neurons. We assessed the contribution of C9ORF72 to MN degeneration by knocking out the start codon and comparing the transcriptome.
创建时间：
2020-04-21
