Exogenous SLFN12 reduces in vivo TNBC proliferation and invasion, increases luminal markers and a gene signature that reducts better breast cancer survival in cohort database analysis
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE215442
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Our results suggest that increased SLFN12 expression modulates TNBC aggressiveness through a gene signature which could offer new targets for treatment. Human TNBC tumor xenografts with exogenous SLFN12 expression were created and the growth was observed and then RNA-seq was performed to investigate pathways related to SLFN12
创建时间：
2023-01-26
