首页/数据集/Chemical inhibition of Histone Deacetylases 1 and 2 induces fetal hemoglobin through activation of Gata2. Chemical inhibition of Histone Deacetylases 1 and 2 induces fetal hemoglobin through activation of Gata2

Chemical inhibition of Histone Deacetylases 1 and 2 induces fetal hemoglobin through activation of Gata2. Chemical inhibition of Histone Deacetylases 1 and 2 induces fetal hemoglobin through activation of Gata2

NIAID Data Ecosystem2026-03-09 收录

表观遗传治疗

血红蛋白疾病机制

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA259549 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries GSE60791 and GSE60792. Overall design: Refer to individual Series

应用场景：

创建时间：

2014-08-26

癌症分子机制

表观遗传治疗

Nupr1 is a chromatin protein which cooperates with KrasG12D to induce PanIN formation in mice, though the molecular mechanisms underlying this effect remain to be fully characterized. In the current s

NIAID Data Ecosystem70

Editing DNA Methylation in vivo [WGBS]

DNA甲基化

表观遗传治疗

DNA methylation is an epigenetic mechanism essential for orchestrating gene expression. Precise editing of DNA methylation has emerged as a new method to dissect its biological function in a DNA seque

NIAID Data Ecosystem30

Inducible mucosa-like differentiation of head and neck cancer cells drives the epigenetically determined loss of cell malignancy. Inducible mucosa-like differentiation of head and neck cancer cells drives the epigenetically determined loss of cell malignancy

头颈部鳞状细胞癌分子分型

表观遗传治疗

Human papillomavirus-negative head and neck squamous cell carcinoma (HNSCC) is a highly malignant disease with high death rates that have remained substantially unaltered for decades. Therefore, new t

NIAID Data Ecosystem10

Supplementary Table S2 from Association of DNA Methylation of Phosphoserine Aminotransferase with Response to Endocrine Therapy in Patients with Recurrent Breast Cancer

癌症生物标志物

表观遗传治疗

Supplementary Table S2 from Association of DNA Methylation of Phosphoserine Aminotransferase with Response to Endocrine Therapy in Patients with Recurrent Breast Cancer

Figshare2005-05-15 更新60

Gene expression profiles of T-cell acute lymphoblastic leukemia cell lines with and without chronic GSI-treatment. Homo sapiens

白血病耐药机制

表观遗传治疗

Here we modeled T-ALL resistance to Notch inhibition, identifying ‘persister’ cells that readily expand in the presence of gamma secretase inhibitor (GSI) and the absence of Notch signaling. Rare pers

NIAID Data Ecosystem20

Chemical inhibition of Histone Deacetylases 1 and 2 induces fetal hemoglobin through activation of Gata2. Chemical inhibition of Histone Deacetylases 1 and 2 induces fetal hemoglobin through activation of Gata2

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