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Gene expression of MOLM14 (AML cell line) transfected with siATF4 or siCTRL

NIAID Data Ecosystem2026-03-12 收录

白血病分子机制

基因沉默转录组学

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE151130 数据链接链接失效反馈

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资源简介：

Assessment of the impact of ATF4 silencing in FLT3-ITD/MLL AML cell lines siATF4 versus siCTL MOLM14 transcriptome

应用场景：

创建时间：

2021-05-25

白血病分子机制

肿瘤抑制基因

Lentiviral constructs expressing candidate tumoure suppressor genes were transduced into 697 cells. Of the candidates FOXO3, two isoforms of PRDM1 (⍺ and β) suppressed growth in-vitro and in-vivo. To

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ZFP36L2 post-transcriptionally controls acute myeloid leukemia differentiation (MOLM13 HiC). ZFP36L2 post-transcriptionally controls acute myeloid leukemia differentiation (MOLM13 HiC)

白血病分子机制

三维基因组学

Genome-wide assessment of TADs in AML cells Overall design: TAD domains in MOLM13 AML cells

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mCG-PML-RARA alters multipotent hematopoiesis.

造血表观遗传学

白血病分子机制

mCG-PML-RARA alters multipotent hematopoiesis.

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Gene expression profile of the human T-ALL cell line JURKAT after TYK2 and STAT1 knockdown

白血病分子机制

基因靶向治疗

Targeted molecular therapy has yielded remarkable outcomes in certain cancers, but specific therapeutic targets remain elusive for many others. As a result of two independent RNA interference (RNAi) s

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Interaction of c-Myb with p300 is required for the induction of acute myeloid leukemia (AML) by human AML oncogenes

白血病分子机制

致癌蛋白相互作用

Interaction of c-Myb with p300 is required for the induction of acute myeloid leukemia (AML) by human AML oncogenes

ChEBI2012-12-31 更新00