首页/数据集/Supplementary Table 1 from RhoJ Regulates Melanoma Chemoresistance by Suppressing Pathways That Sense DNA Damage

Supplementary Table 1 from RhoJ Regulates Melanoma Chemoresistance by Suppressing Pathways That Sense DNA Damage

NIAID Data Ecosystem2026-05-16 收录

数据链接：

https://figshare.com/articles/dataset/Supplementary_Table_1_from_RhoJ_Regulates_Melanoma_Chemoresistance_by_Suppressing_Pathways_That_Sense_DNA_Damage/22395875 数据链接链接失效反馈

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资源简介：

XLS file - 9MB, genome-wide RNAi dataset

应用场景：

创建时间：

2012-11-01

黑色素瘤耐药性

RAS突变肿瘤

Targeted therapies have the potential to revolutionize cancer care by providing personalized treatment strategies that are less toxic and more effective but it is clear that for most solid tumors supp

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Transcriptome analysis of wild-type and drug-resistance-induced melanoma cell lines

黑色素瘤耐药性

转录组学治疗

Transcriptional co-acticvators inhibition is proving to be a powerful mean against cancer cells that shown a novel hallmarks named transcriptional addiction. This dependecy on co-activars activity lay

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Additional file 3 of Diverse mechanisms activate the PI 3-kinase/mTOR pathway in melanomas: implications for the use of PI 3-kinase inhibitors to overcome resistance to inhibitors of BRAF and MEK

黑色素瘤耐药性

PI3K磷脂酰肌醇3-激酶

Additional file 3: Supplementary Table S3: Variants of genes encoding class I PI3K regulatory subunits

Figshare2021-02-06 更新30

Proteomics identifies markers of MAPKi resistance offering new therapeutic strategies in melanoma- Cytoplasmic (cyt), nuclear (ne) and secreted (sn) proteins of MAPKi sensitive melanoma cells

黑色素瘤耐药性

蛋白质组学生物标志物

MAPK inhibitors (MAPKi) show outstanding clinical response rates in melanoma patients harbouring BRAF mutations, but resistance is common. High-throughput, subcellular proteome analyses at two differe

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Acetylcytidine modification of DDX41 and ZNF746 by N-acetyltransferase 10 contributes to chemoresistance of melanoma

黑色素瘤耐药性

RNA修饰机制

We performed RNA-seq to analyse the different expression genes of DTIC-resistant (DR) A375 cells and wild-type control. Examination of differentially expressed genes in DR A375 cells compared with wil

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Supplementary Table 1 from RhoJ Regulates Melanoma Chemoresistance by Suppressing Pathways That Sense DNA Damage

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