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A primary cell wall cellulose-dependent defense mechanism against vascular pathogens revealed by time-resolved dual-transcriptomics

NIAID Data Ecosystem2026-03-12 收录

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病原-宿主互作

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE168919 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

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创建时间：

2021-08-23

辐射预测

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Analysis of human peripheral blood 48 hours after irradiation ex vivo with graded doses of gamma rays. Results have been used in building and testing classifiers to predict exposure dose for use in ra

NIAID Data Ecosystem90

Crystal structure of NN domain of resistance protein

植物抗病蛋白

病原-宿主互作

Crystal structure of NN domain of resistance protein Descriptor: Tm-1 protein Authors: Matsumura, H, Katoh, E. Deposit date: 2014-02-27 Release date: 2014-08-13 Last modified: 2024-03-20 Method: X-RAY

Protein Data Bank Japan2024-03-20 更新60

Expression data from C. elegans. Caenorhabditis elegans

病原-宿主互作

基因表达调控

We exposed wild-type Vibrio cholerae E7496, multiple Vibrio cholerae virulence factor deleted genes with intact hemolysin A gene [CVD109] and without hemolysin A gene [CVD110] in E7946, and E.coli OP5

NIAID Data Ecosystem20

High-throughput discovery of human antimicrobial peptide activators of Salmonella Typhimurium PhoPQ

抗菌肽筛选

病原-宿主互作

We developed a surface-displayed library of 117 human antimicrobial peptides (AMPs) and over 3000 human AMP variants in a laboratory E. coli strain expressing PhoP and PhoQ from Salmonella Typhimurium

NIAID Data Ecosystem80

Transcriptome and proteome analysis of Salmonella enterica serovar Typhimurium systemic infection of wild type and immune-deficient mice

病原-宿主互作

疾病多组学分析

Salmonella enterica are a threat to public health. Current vaccines are not fully effective. The ability to grow in infected tissues within phagocytes is required for S. enterica virulence in systemic

Figshare2017-08-11 更新20

A primary cell wall cellulose-dependent defense mechanism against vascular pathogens revealed by time-resolved dual-transcriptomics

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