Analysis of HER2-amplified NCI-N87 gastric cancer cell line exposed to 0.1 µg/mL T-DM1 and subsequently cultured in the presence of gradually increasing doses, up to a maximum of 4 µg/mL. The resultan

NIAID Data Ecosystem20

Additional file 1: of Tunicamycin specifically aggravates ER stress and overcomes chemoresistance in multidrug-resistant gastric cancer cells by inhibiting N-glycosylation

肿瘤耐药机制

药物敏感性

Table S1. CI values for SGC7901, SGC7901/ADR and SGC7901/VCR. CI was calculated by CompuSyn, which is based on the model created by Chou and Talalay. CI 1 represent synergism, add

NIAID Data Ecosystem20

Suppression of adaptive responses to targeted cancer therapy by transcriptional repression [RNA-seq]. Homo sapiens

肿瘤耐药机制

转录组药物响应

Large-scale genomic profiling efforts have facilitated the characterization of molecular alterations in cancers and aided the development of targeted kinase inhibitors for a wide array of cancer types

NIAID Data Ecosystem50

Fusion genes in EGFR mutant lung cancer

肿瘤耐药机制

融合基因验证

The clinical significance of gene fusions detected by DNA-based next generation sequencing remains unclear as resistance mechanisms to EGFR tyrosine kinase inhibitors (TKIs) in EGFR mutant non-small c

NIAID Data Ecosystem20

Engineering ABT-737 Resistance in MYC-driven Lymphomas Identifies DHX9 as a Drug Response Modifier

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