Pulmonary arterial hypertension (PAH) is a progressive disease characterized by elevated pulmonary arterial pressures that can lead to right heart failure and death. No cure exists for this disease, b

NIAID Data Ecosystem10

Nuclear Factor Kappa B is central to MarekM-bM-^@M-^Ys Disease lymphomagenesis

禽类肿瘤机制

核因子κB信号通路

Nuclear Factor Kappa B is central to MarekM-bM-^@M-^Ys Disease lymphomagenesis

ChEBI2012-03-15 更新00

SNP rs9277336 Controls the ACTN4-HLA-DPA1 Axis and Pulmonary Endothelial Pathophenotypes in Pulmonary Arterial Hypertension. SNP rs9277336 Controls the ACTN4-HLA-DPA1 Axis and Pulmonary Endothelial Pathophenotypes in Pulmonary Arterial Hypertension

肺动脉高压分子病理

基因调控网络

HLA-DPA1-related SNP rs9277336 is linked to increased PAH risk and binds ACTN4 in an allele-specific fashion. HLA-DPA1 transcript is decreased in PAH lung endothelial cells and controlled by ACTN4. AC

NIAID Data Ecosystem30

Molecular changes implicate angiogenesis and arterial remodeling in systemic sclerosis-associated and idiopathic pulmonary hypertension

肺动脉高压分子病理

单细胞转录组学

Pulmonary hypertension (PH) is a common complication of systemic sclerosis (SSc) and a leading cause of mortality among patients with this disease. PH can also occur as an idiopathic condition (idiopa

NIAID Data Ecosystem00

File S1 - Nuclear Factor κ-B Is Activated in the Pulmonary Vessels of Patients with End-Stage Idiopathic Pulmonary Arterial Hypertension

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