KDM5B ChIP-Seq project. Mus musculus
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下载链接：
https://www.ncbi.nlm.nih.gov/bioproject/PRJNA270238
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资源简介：
Identify binding sites and epigenetic regulation of KDM5B histone demethylase
应用场景：
创建时间：
2014-12-12
相关数据集
KDM5B ChIP-Seq project
Identify binding sites and epigenetic regulation of KDM5B histone demethylase
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The KDM5B histone H3 lysine 4 (H3K4) demethylase has been implicated in therapy resistance in multiple cancer types including breast cancer, but the underlying mechanism is poorly defined. Here we sho
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Extended self-renewal and accelerated reprogramming in the absence of Kdm5b [ChIP-Seq]. Mus musculus
ES cell pluripotency is thought to be regulated in part by H3K4 methylation. However, it is unclear how H3K4 demethylation contributes to ES cell function and participates in iPS cell reprogramming. H
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Kdm5b depletion in embryonic stem cells does not induce intragenic promoters or affect transcription [ChIP-Seq]. Mus musculus
The application of massively-parallel sequencing (MPS) technology to understanding how the genome is regulated is of major importance, as we now appreciate sequence variants associated with common her
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