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资源简介：
the Pan-Cancer Role of HDAC2
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创建时间：
2025-04-15
相关数据集
Control ChIP-seq from HCT116 (ENCSR936ENS)
For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for_External_Users_03-07-14.pdf https://www.e
NIAID Data Ecosystem60
Prevention of chromatin destabilization by FACT is crucial for malignant transformation. Prevention of chromatin destabilization by FACT is crucial for malignant transformation
Histone chaperone FACT is commonly expressed and essential for the viability of transformed but not normal cells and its expression levels correlate with poor prognosis in cancer patients. FACT binds
NIAID Data Ecosystem40
H3K4 demethylase KDM5B regulates epigenetic plasticity and cancer cell identity [RNA-seq]. H3K4 demethylase KDM5B regulates epigenetic plasticity and cancer cell identity [RNA-seq]
The H3K4 demethylase KDM5B is overexpressed in multiple cancer types, but the underlying mechanistic contribution of dysregulated H3K4 demethylation in cancer is poorly understood. Here, we show that
NIAID Data Ecosystem20
Supplementary Table S4 from Discovery of Candidate DNA Methylation Cancer Driver Genes
Copy number changes in MethSig CLL DNAme drivers.
Figshare2021-09-01 更新50
DNA methylation analysis of esophageal squamous cell carcinoma
Upper aerodigestive tract (UADT) tumors present different biological behavior and prognosis, suggesting specific molecular mechanisms underlying their development. However, they are rarely considered
NIAID Data Ecosystem30
