官方服务:
资源简介:
Knock out of NOTCH1-bound MYC putative enhancer (N-Me)
应用场景:
创建时间:
2014-09-07
相关数据集
TF ChIP-seq from K562 (ENCSR350GZC)
eGFP-ZNF561 ChIP-seq on human K562 For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for_Exter
NIAID Data Ecosystem60
The Myc binding partner Spt5 is essential for brain tumor growth in Drosophila. The Myc binding partner Spt5 is essential for brain tumor growth in Drosophila
The transcription factor SPT5 physically interacts with MYC oncoproteins and is essential for the full transcriptional activation of MYC targets in cultured cells. Here, we use Drosophila to show that
NIAID Data Ecosystem50
Figure S1-4 and Table S1-2
Mechanistic Insights into SENP1 and OCT4 Interaction in Promoting Drug Resistance and Stem Cell Features in Colon CancerFigure S1-4 and Table S1-2
Figshare2025-01-22 更新40
Transcriptome Analysis of RUVBL1
RUVBL1 was shown to be essential for tumor cell growth of many cancers.We identified a lot of genes that are changed obviously by RUVBL1. Gene ontology and Kyoto Encyclopedia of Genes and Genomes anal
NIAID Data Ecosystem60
CD95/Fas increases stemness in cancer cells by inducing a STAT1 dependent Type I interferon response [array]
CD95 was found to be important for both the formation of CSCs and their maintenance. Using a stable isotope labeling by amino acids in cell culture (SILAC) analysis we have now identified STAT1 as a
NIAID Data Ecosystem40



