PRC2 controls adult neuron identity and protects neurons against neurodegeneration
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PRC2 controls adult neuron identity and protects neurons against neurodegeneration
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创建时间：
2016-07-29
相关数据集
Acetyl CoA metabolism by ACSS2 regulates neuronal histone acetylation and hippocampal memory [RNA-seq In Vivo]
Metabolic production of acetyl-CoA has been linked to histone acetylation and gene regulation, however the mechanisms are largely unknown. We show that the metabolic enzyme acetyl-CoA synthetase 2 (AC
NIAID Data Ecosystem100
Active N6-Methyladenine demethylation by DMAD regulates gene expression by coordinating with Polycomb protein in neurons
Raw images labeled as shown in the main and supplementary figures. The actual regions used in the figures were highlighted by red frame.
NIAID Data Ecosystem70
PRC2-mediated repression is essential to maintain identity and function of differentiated dopaminergic and serotonergic neurons [ChIP-Seq]. PRC2-mediated repression is essential to maintain identity and function of differentiated dopaminergic and serotonergic neurons [ChIP-Seq]
Neurons of the CNS must maintain their distinct identity over an entire lifespan. Apart from instructive information provided by transcription factors driving neuron specific genes, other gene program
NIAID Data Ecosystem40
Active N6-Methyladenine demethylation by DMAD regulates gene expression by modulating the binding dynamics of Polycomb protein in neurons
A Ten-Eleven Translocation (TET) ortholog has been identified as a DNA N6-Methyladenine (6mA) demethylase (DMAD) in Drosophila. However, the molecular roles of 6mA and DMAD remain largely unexplored.
NIAID Data Ecosystem80
Acetyl CoA metabolism by ACSS2 regulates neuronal histone acetylation and hippocampal memory [RNA-seq In Vitro]
Metabolic production of acetyl-CoA has been linked to histone acetylation and gene regulation, however the mechanisms are largely unknown. We show that the metabolic enzyme acetyl-CoA synthetase 2 (AC
NIAID Data Ecosystem40
